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DH has plaque in his coronary arteries - now what? UPDATE #26 - Page 2

post #21 of 52
Originally Posted by TanyaLopez View Post
The plaque is calcium, right? I've read studies that talk about K2 and calcium utilization, the vitamin K yahoo group has a Files section that has a lot of studies on K2. The yahoo group is about kids with autism, but anyone can join and they've got a nice collection of articles, and I thought there was some heart health/calcium utilization stuff there. Some of the WAPF articles might have citations on heart stuff and K2 and you can follow them back to the original studies and see what's up.

I could also be wrong about this, but I'd read about folate and the MTHFR gene mutation. I thought early heart problems were one problem associated with a variant of MTHFR. Some people need more dietary folate than others, 800mcg vs the 400mcg for most of us, and folic acid doesn't work the same way as folate (because of this gene--for many of us, folic acid is fine). I could be remembering wrong, but I'd do some reading on this. There is a genetic test for it if the doc is willing to do it. Google aspirin and MTHFR as well, because I think aspirin is involved, but it does things somewhat backward.
I think part of the connectionwith MTHFR and folic acid has to do with homocysteine. Researchers are seeing that homocysteine levels are hugely important for heart health - more so than cholesterol. A google search will bring things up. Folic acid and a couple of other things will bring down homocysteine levels.
post #22 of 52
I just sent you a PM about statin risks, vit D and folate benefits:
Memory loss
, http://www.ivanhoe.com/channels/p_ch...?storyid=10491

Blocks CoQ10 production. CoQ10 is a coenzyme necessary for the production of ATP (adenosine triphosphate). ATP is the source for cellular energy within the human heart. As CoQ10 is diminished, the heart weakens. Over time, this can result in congestive heart failure (CHF).

Liver damage, muscle pain:

: http://www.medscape.com/viewarticle/496431_4

peripheral neuropathy, or pain or numbness in the extremities like fingers and toes: http://seniorjournal.com/NEWS/Health...rchersFind.htm

"Statin-associated peripheral neuropathy may persist for months or years after withdrawal of the statin. In two ADRAC cases of persistent peripheral neuropathy, motor and sensory conduction tests showed minimal recovery 4 and 12 months, respectively, after discontinuation of simvastatin, despite clinical improvement. A further 21 cases had not recovered at the time of reporting, between one and eight months after discontinuation of the statin. In two other reports, the problem was persisting after 3 and 5 years, respectively." http://www.procor.org/research/resea...?doc_id=982615

cancer risk: http://www.cancerdecisions.com/conte.../lang,english/

Statins can also cause your CPK levels to be mildly elevated.

Strokes: The cholesterol-lowering medication atorvastatin (Lipitor) may slightly increase the risk of hemorrhagic stroke, or bleeding in the brain, when taken by people who have already had a stroke, according to a Duke University Medical Center researcher. But it also reduces the risk of having a second stroke or coronary heart event.


Cholesterol Is Not A Major Cause Of Arterial Disease

Several factors appear to be of greater importance than cholesterol in causing arterial disease. Among these are deposition of toxic metals in the lining endothelium of arteries, Vitamin C deficiency, excessive amounts of lipoprotein (a), inflammation in arteries, excessive clotting of blood, homocysteine elevation (hyperhomocystinemia) and dangerous foods.

An important study by Dr. Harlan Krumholz revealed that persons with low cholesterol levels over the age of 70 died twice as often from heart attacks[3] as older persons with high cholesterol values. Most studies in old persons have shown that cholesterol is not a risk factor for coronary artery disease. Approximately 90 % of cardiovascular disease is seen in persons over 60 years of age. Almost all studies have shown that high cholesterol is not a risk factor for women.[4] This leaves cholesterol as a risk factor for less than 5 % of those persons dying of a heart attack.

High cholesterol values protect against infection. In a review of 19 studies involving 68,000 persons low cholesterol values revealed an increased risk for dying from lung and gastrointestinal diseases. Both lung and g.i. diseases are often related to infections.


Benefits of High Cholesterol:

1. Krumholz HM and others. Lack of association between cholesterol and coronary heart disease mortality and morbidity and all-cause mortality in persons older than 70 years. Journal of the American Medical Association 272, 1335-1340, 1990.

2. Ravnskov U. High cholesterol may protect against infections and atherosclerosis. Quarterly Journal of Medicine 96, 927-934, 2003.

3. Jacobs D and others. Report of the conference on low blood cholesterol: Mortality associations. Circulation 86, 1046–1060, 1992.

4. Iribarren C and others. Serum total cholesterol and risk of hospitalization, and death from respiratory disease. International Journal of Epidemiology 26, 1191–1202, 1997.

5. Iribarren C and others. Cohort study of serum total cholesterol and in-hospital incidence of infectious diseases. Epidemiology and Infection 121, 335–347, 1998.

6. Claxton AJ and others. Association between serum total cholesterol and HIV infection in a high-risk cohort of young men. Journal of acquired immune deficiency syndromes and human retrovirology 17, 51–57, 1998.

7. Neaton JD, Wentworth DN. Low serum cholesterol and risk of death from AIDS. AIDS 11, 929–930, 1997.

8. Rauchhaus M and others. Plasma cytokine parameters and mortality in patients with chronic heart failure. Circulation 102, 3060-3067, 2000.

9. Niebauer J and others. Endotoxin and immune activation in chronic heart failure. Lancet 353, 1838-1842, 1999.

10. Vredevoe DL and others. Skin test anergy in advanced heart failure secondary to either ischemic or idiopathic dilated cardiomyopathy. American Journal of Cardiology 82, 323-328, 1998.

11. Rauchhaus M, Coats AJ, Anker SD. The endotoxin-lipoprotein hypothesis. Lancet 356, 930–933, 2000.

12. Rauchhaus M and others. The relationship between cholesterol and survival in patients with chronic heart failure. Journal of the American College of Cardiology 42, 1933-1940, 2003.

13. Horwich TB and others. Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure. Journal of Cardiac Failure 8, 216-224, 2002.

14. Elias ER and others. Clinical effects of cholesterol supplementation in six patients with the Smith-Lemli-Opitz syndrome (SLOS). American Journal of Medical Genetics 68, 305–310, 1997.

15. Bhakdi S and others. Binding and partial inactivation of Staphylococcus aureus a-toxin by human plasma low density lipoprotein. Journal of Biological Chemistry 258, 5899-5904, 1983.

16. Flegel WA and others. Inhibition of endotoxin-induced activation of human monocytes by human lipoproteins. Infection and Immunity 57, 2237-2245, 1989.

17. Weinstock CW and others. Low density lipoproteins inhibit endotoxin activation of monocytes. Arteriosclerosis and Thrombosis 12, 341-347, 1992.

18. Muldoon MF and others. Immune system differences in men with hypo- or hypercholesterolemia. Clinical Immunology and Immunopathology 84, 145-149, 1997.

19. Feingold KR and others. Role for circulating lipoproteins in protection from endotoxin toxicity. Infection and Immunity 63, 2041-2046, 1995.

20. Netea MG and others. Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections. Journal of Clinical Investigation 97, 1366-1372, 1996.

21. Harris HW, Gosnell JE, Kumwenda ZL. The lipemia of sepsis: triglyceride-rich lipoproteins as agents of innate immunity. Journal of Endotoxin Research 6, 421-430, 2001.

22. Netea MG and others. Hyperlipoproteinemia enhances susceptibility to acute disseminated Candida albicans infection in low-density-lipoprotein-receptor-deficient mice. Infection and Immunity 65, 2663-2667, 1997.

23. Ross R, Glomset JA. The pathogenesis of atherosclerosis. New England Journal of Medicine 295, 369-377, 1976.

24. Ross R. The pathogenesis of atherosclerosis and update. New England Journal of Medicine 314, 488-500, 1986.

25. Klotz O, Manning MF. Fatty streaks in the intima of arteries. Journal of Pathology and Bacteriology. 16, 211-220, 1911.

26. At least 200 reviews about the role of infections in atherosclerosis and cardiovascular disease have been published; here are a few of them: a) Grayston JT, Kuo CC, Campbell LA, Benditt EP. Chlamydia pneumoniae strain TWAR and atherosclerosis. European Heart Journal Suppl K, 66-71, 1993. b) Melnick JL, Adam E, Debakey ME. Cytomegalovirus and atherosclerosis. European Heart Journal Suppl K, 30-38, 1993. c) Nicholson AC, Hajjar DP. Herpesviruses in atherosclerosis and thrombosis. Etiologic agents or ubiquitous bystanders? Arteriosclerosis Thrombosis and Vascular Biology 18, 339-348, 1998. d) Ismail A, Khosravi H, Olson H. The role of infection in atherosclerosis and coronary artery disease. A new therapeutic target. Heart Disease 1, 233-240, 1999. e) Kuvin JT, Kimmelstiel MD. Infectious causes of atherosclerosis. f.) Kalayoglu MV, Libby P, Byrne GI. Chlamydia pneumonia as an emerging risk factor in cardiovascular disease. Journal of the American Medical Association 288, 2724-2731, 2002.

27. Grau AJ and others. Recent bacterial and viral infection is a risk factor for cerebrovascular ischemia. Neurology 50, 196-203, 1998.

28. Mattila KJ. Viral and bacterial infections in patients with acute myocardial infarction. Journal of Internal Medicine 225, 293-296, 1989.

29. The successful trials: a) Gurfinkel E. Lancet 350, 404-407, 1997. b) Gupta S and others. Circulation 96, 404-407, 1997. c) Muhlestein JB and others. Circulation 102, 1755-1760, 2000. d) Stone AFM and others. Circulation 106, 1219-1223, 2002. e) Wiesli P and others. Circulation 105, 2646-2652, 2002. f) Sander D and others. Circulation 106, 2428-2433, 2002.

30. The unsuccessful trials: a) Anderson JL and others. Circulation 99, 1540-1547, 1999. b) Leowattana W and others. Journal of the Medical Association of Thailand 84 (Suppl 3), S669-S675, 2001. c) Cercek B and others. Lancet 361, 809-813, 2003. d) O’Connor CM and others. Journal of the American Medical Association. 290, 1459-1466, 2003.

31. Gieffers J and others. Chlamydia pneumoniae infection in circulating human monocytes is refractory to antibiotic treatment. Circulation 104, 351-356, 2001

32. Gurfinkel EP and others. Circulation 105, 2143-2147, 2002.


Vitamin D is Synthesized From Cholesterol and Found in Cholesterol-Rich Foods

One of cholesterol's many functions in the body is to act as a precursor to vitamin D.

Vitamin D can also be obtained from foods. Interestingly, foods that provide this vitamin -- all of which are animal foods -- tend to be high in cholesterol.

Since cholesterol is a precursor to vitamin D, inhibiting the synthesis of cholesterol will also inhibit the synthesis of vitamin D. Since sunlight is required to turn cholesterol into vitamin D, avoiding the sun will likewise undermine our ability to synthesize vitamin D. And since vitamin D-rich foods are also rich in cholesterol, low-cholesterol diets are inherently deficient in vitamin D. http://www.cholesterol-and-health.com/Vitamin-D.html


The Right Fats

The assimilation and utilization of vitamin D is influenced by the kinds of fats we consume. Increasing levels of both polyunsaturated and monounsaturated fatty acids in the diet decrease the binding of vitamin D to D-binding proteins. Saturated fats, the kind found in butter, tallow and coconut oil, do not have this effect. Nor do the omega-3 fats.66 D-binding proteins are key to local and peripheral actions of vitamin D. This is an important consideration as Americans have dramatically increased their intake of polyunsaturated oils (from commercial vegetable oils) and monounsaturated oils (from olive oil and canola oil) and decreased their intake of saturated fats over the past 100 years.

In traditional diets, saturated fats supplied varying amounts of vitamin D. Thus, both reduction of saturated fats and increase of polyunsaturated and monounsaturated fats contribute to the current widespread D deficiency.

Trans fatty acids, found in margarine and shortenings used in most commercial baked goods, should always be avoided. There is evidence that these fats can interfere with the enzyme systems the body uses to convert vitamin D in the liver.80


Debunking the Cholesterol Myth:


We’re lead to believe that cholesterol is an enemy, particularly to cardiovascular health. But think about it, why does your liver naturally produce something that’s detriment to your health? It doesn’t.

Through tapes on the raft, the lipid hypothesis reigns supreme. It dictates that in order to stay healthy you should reduce your cholesterol (to an ever receding value) by consuming a low fat diet and exercise (no prob with latter though!). And If that fails you should try controversial medicines called statins.

They are notable for alarming side effects.

Sounds nice and simple? In England 2003, the average cholesterol value was 6.1, in France it was 6.2. The recommended cholesterol level is < 5mmol. This means a lot of people ’suffer’ from a symptomless disease. But here’s the best bit: our local French neighbours don’t suffer from heart disease much, definitely not as much as us. In Japan (2005), the level of cholesterol went up as their rate of heart disease dropped, perhaps because their general lifestyle is better than the Brits to avoid inflammation of the heart in the first place despite possible low vit D. Populations with the least level of cholesterol saw higher rates of heart disease. But furthermore, high cholesterol for women the world over is not a risk factor. Are they really that different?

Did you know that vitamin D is created from cholesterol? Provide your skin with adequate sunlight and excess cholesterol is turned into vitamin D, a hormone with responsibilities including calcium metabolisation, blood sugar regulation, blood pressure regulation, mental health function, diabetes prevention…

And if your excess cholesterol isn’t turned into D, it accumulates doesn’t it?…I can find no answers, at least online that refutes or proves this. Therefore could high cholesterol just be a signifier of low vitamin D, a hormone that is said to have anti-inflammatory properties?…


Retrospective studies: When the men were grouped according to vitamin D intake above and below 2.5 mug and serum cholesterol levels above or below 250 mg %, a significant relationship appeared (using Yates's correction X2=10.3, P=0.0013 and the correlation coefficient 0.011 less than P less than 0.005). http://www.ncbi.nlm.nih.gov/pubmed/1179189

Effects of Atorvastatin on vitamin D levels in patients with acute ischemic heart disease.:
In conclusion, atorvastatin increases vitamin D levels. This increase could explain some of the beneficial effects of atorvastatin at the cardiovascular level that are unrelated to cholesterol levels. http://www.ncbi.nlm.nih.gov/pubmed/17398180

It ain't lowering cholesterol (which the liver regulates anyway) which lowers heart risk, from my reading. There are many reasons why the dietary-heart-cholesterol hypothesis should be questioned, and why statins might be acting in some other way to reduce the risk of coronary heart disease. Here, I propose that rather than being cholesterol-lowering drugs per se, statins act as vitamin D analogues, and explain why. This proposition is based on published observations that the unexpected and unexplained clinical benefits produced by statins have also been shown to be properties of vitamin D. It seems likely that statins activate vitamin D receptors. http://www.ncbi.nlm.nih.gov/pubmed/16815382

Cholesterol: Friend Or Foe?

How Do High-Folate Diets Protect Against Heart Disease? http://www.medicalnewstoday.com/articles/40413.php


Folate Consumption Reduces Risk of Stroke by 20 Percent in Male Smokers http://www.naturalnews.com/025324_st...late_food.html

Folate, brain health and stroke: http://www.naturalnews.com/023134_fo...ocysteine.html
and allergies: http://www.naturalnews.com/026225_fo...allergies.html

post #23 of 52
Thread Starter 
By the way, I just want to say - MDC rocks!!! It's been a while since I've come here for any real advice - guess the little ones are past the sleep/eat/nursing/behavior problems stage and I am just so touched and awed at the response. Incredible info. Thank you everyone! (keep it coming if you've got it! I'm deep-diving into researching this and all the links provided)
post #24 of 52
You may want to read the South Beach Diet book. In it, the doctor/author talks about how the diet was developed for heart health, not for losing weight (that was a side effect). Anyway, it's basically another vote for a low-carb + good fats diet. He also advocates the statin/aspirin combo.
post #25 of 52
CoQ10 supplement is necessary for heart problems, also what Chakra said. Has he tried to increase the amount of raw foods he eats? Has he tried juicing?
post #26 of 52
Thread Starter 

I wish it were a better update. DH's lab results came in today. Here were the values of interest (brace yourselves):

(by the way, this was a FASTING blood test, taken 12 hours after last meal)

GLUCOSE 101 (reference range: 74-99 mg/dL)
CHOLESTEROL 230 (reference range: 100-200mg/dL)
HDL 46 (reference range: 40-59 mg/dL)
LDL 172 (reference range: 0-129 mg/dL)
THYROID 1.19 (reference range: 0.27-4.20 uIU/mL)

Deep breath.

Ok, I think the glucose is actually high. I mean, it's only 2 above the reference range, and his doctor didn't mention it in his write up, but a fasting glucose of 101 makes me think DH should probably get a full workup for Type II Diabetes or at least pre-diabetes or whatever. I know there is a connection between blood sugar levels and artherosclerosis, but I'm not really clear on exactly how that works. Still reading on that one. Anyone have any more info? By comparison, my latest fasting glucose was 78 and it's never been above 85/86.

Cholesterol. Holy crap. His cholesterol has never ever been above 200 before. And now his LDL is so out of whack. LDL of 172!!!! And HDL (good cholesterol) is a pitiful 46. This is scary. In fact, these numbers are so bad it makes me re-think the statin advice. (His doctor wants him on 40mg/day of SIMVASTATIN starting immediately. Up until now I felt this was nuts because we weren't expecting his cholesterol to actually be this high (and the ratio so horrible).

Thyroid. My feeling is that even though this is well within the reference range, that this thyroid number is sub-optimal. Can any of our thyroid experts here weigh in on this?
post #27 of 52
You'd need free-T3 and free-T4 to know what's going on, that TSH alone isn't very helpful unless he doesn't have any symptoms that point to thyroid.

With the family history you've discussed, I think changes are in order, but if I had total cholesterol of 230, I wouldn't mind. Beats the heck out of 130 (actually 138, which is where I was last year). But based on his family history and the fasting glucose, changes are in order.

I think you've got a lot of good stuff to read here. For us (DH and I have had health issues in the past few years) we're focusing on figuring out what _our_ bodies need, and what we are less tolerant of, compared to everyone else. There really is a difference in what people need and can tolerate (in the healthy sense, not in the "surely Cokes and Twinkies every day isn't _too_bad_, is it?" way). Our bodies can work well, if we provide enough of the good stuff and reduce the bad.
post #28 of 52
Did they run a fasting insulin? That would be interesting.

I'm not at all concerned with the 230 cholesterol - it's the ratio that is really alarming.

Your DH REALLY has to get on board with diet changes. He needs to eat like a diabetic - like a diabetic *really* should, not like the diabetic association recommends. Bernstein lays this all out in his book. Cutting back on carbs & sugar will help with the blood sugar/insulin problems which lead to heart disease.

If it were my loved one, I'd drastically change the diet - & be hardcore about it. That's all I'd do for now. Lots of good fats, good animal protein, non-starchy veggies. No fruit or grains or starchy veggies. (I think low sugar berries, like blueberries, raspberries, etc) would be okay here & there.)
Then I'd recheck the cholesterol & blood glucose & blood insulin. I think a challenge is a better way to check how the body responds to a typical meal. Eating a "balanced" meal & then checking insulin an hour or two later would be more telling. Obviously, a meal including a good amount of carbs is going to result in a higher reading than a low carb meal.

Meals should be simple: meat, non-starchy veggies, fat. No snacking between meals. If he gets enough protein & fat in his 3 daily meals, he shouldn't be hungry in between. The body needs *at least* 4 hours to stabilize insulin levels before more food is eaten. Once his body heals, he'll probably be able to snack again, but nothing carby/sugary.
post #29 of 52
Thread Starter 
Metasequoia... slightly OT - what about the popular dieting advice to eat 6 small meals a day or whatever? And people on MDC (and some pediatricians/child experts) say you should let kids graze all day - that they eat healthier this way, self-regulate better, etc. Really - the old 3 squares a day advice is best?
post #30 of 52
He might think about going raw...check out the film Raw for 30.
post #31 of 52
I would be most concerned about the HDL being low and the ratio of HDL and LDL.
230 is too high also but if you incorporate changes to increase HDL then you will naturally lower the LDL

From what I've read, statins can actually lower your HDL.

I don't agree with the saturated fat or red meat being good, but increasing what is popularly called "good fats" has worked for me, specifically olive oil, nuts and avocado, along with cutting out trans fat and refined foods.

Exercise is very, very important and it doesn't have to be overly strenuous but aerobic is best several times a week.

Taking fish oil (I take flax seed oil because I'm vegetarian), drinking OJ and cranberry juice, using curcumin to spice your food and drinking red wine are all good too.

If he smokes, stop. HDL will go right up.

I agree with you on the fasting glucose, it's high.

Good luck,

post #32 of 52
Originally Posted by Periwinkle View Post
Metasequoia... slightly OT - what about the popular dieting advice to eat 6 small meals a day or whatever? And people on MDC (and some pediatricians/child experts) say you should let kids graze all day - that they eat healthier this way, self-regulate better, etc. Really - the old 3 squares a day advice is best?
Eating 6 "meals" or snacks throughout the day won't give his body a break from insulin production. His body will constantly be pumping insulin to try to deal with the blood sugar effect from food.

There's another book you need to read - it's called Primal Body, Primal Mind - the author talks about ALL of this - very interesting. She calls the USDA food pyramid the "Feedlot Pyramid." I'm ordering it today.

I've been thinking a lot about how the kids should eat. I know I am doing better eating just 3 meals a day, no snacking, VERY low carb. I can already feel that my hypoglycemia is diminishing. I can go many hours w/o food, w/o feeling shaky, irritable, lightheaded, etc. The kids seem to need a little more carb with meals, but they're okay with not snacking. As long as they get enough animal fat & protein with each meal (& some carb), they're okay until the next meal. I have been carrying around just in case snacks for them - kippered herring & crispy pecans & butter.

Originally Posted by Mom2M View Post
230 is too high also but if you incorporate changes to increase HDL then you will naturally lower the LDL
Some of the longest-living & healthiest people have very "high" cholesterol. I agree his ratio is awful, but the overall number isn't what's alarming.

Originally Posted by Mom2M View Post
Exercise is very, very important and it doesn't have to be overly strenuous but aerobic is best several times a week.
Actually, for insulin resistance, anaerobic exercise is preferable. Cardio exercise isn't all its cracked up to be. Building muscle by either weight lifting or resistance training increases insulin sensitivity.

Originally Posted by Mom2M View Post
Taking fish oil (I take flax seed oil because I'm vegetarian), drinking OJ and cranberry juice, using curcumin to spice your food and drinking red wine are all good too.
Drinking OJ (or any juice) is one of the worst things a borderline diabetic (or anyone, really) can do. It's pure glucose once it hits our mouths - no different than eating spoonfuls of white sugar. Even red wine is carby & sugary & should be avoided. BUT, if he's out celebrating or something & wants to have *a* drink, it's one of the better choices - much better than beer.
When children drink juice, it increases their risk of developing diabetes substantially - even the mainstream media (news channels) have been talking about this for years now.
post #33 of 52
referring to the aerobic exercise, red wine and OJ, those are recommended for increasing HDL.
Also, you can eat the oranges and use unsweetened cranberry juice.
His low HDL seems more important than the borderline fasting glucose. Maybe it isn't.
post #34 of 52
Originally Posted by Metasequoia View Post
Eating 6 "meals" or snacks throughout the day won't give his body a break from insulin production. His body will constantly be pumping insulin to try to deal with the blood sugar effect from food.
The body is constantly kicking out insulin (usually in intervals) to reach homeostasis. This is why Lantus is so popular right now, people need a basal rate not just a prandial dose.
post #35 of 52
Originally Posted by Mom2M View Post
referring to the aerobic exercise, red wine and OJ, those are recommended for increasing HDL.
Also, you can eat the oranges and use unsweetened cranberry juice.
His low HDL seems more important than the borderline fasting glucose. Maybe it isn't.
Well, the ultimate concern here is his heart. Fruit is just glucose to his body, it'll spike his blood sugar - then comes all of the insulin & lots of it because there's probably a good deal of insulin resistance going on here. It's insulin resistance that lead to heart failure, not high cholesterol. I agree that his ratio stinks but I think fixing the blood sugar problem is crucial & fruit should be avoided. Once the glucose/insulin is under control, his cholesterol should improve.

Originally Posted by not now View Post
The body is constantly kicking out insulin (usually in intervals) to reach homeostasis. This is why Lantus is so popular right now, people need a basal rate not just a prandial dose.
And the healthy body can moderate insulin at a healthy level, but someone whose body has become insulin resistant due to chronic high blood glucose can't regulate that anymore. The only way to fix it is to allow his body to heal by keeping blood glucose levels as even as possible within a healthy range. This ideal blood glucose level can be different for everyone. But by constantly eating, there's no break.
post #36 of 52
Just found this awesome blog thanks to someone in the TF forum! Here's a great post on carbs & insulin:

The Definitive Guide to Insulin, Blood Sugar & Type 2 Diabetes (and you’ll understand it) BITE ME, ADA

Your DH might like this blog - it's written by a (very fit) guy who has a GREAT sense of humor, very witty & super information. (The one thing wrong with this one post was where he said something about type 2 turning into type 1, but he later acknowledged his slip-up.)
post #37 of 52
Cholesterol in a nutshell. I like this guy too.
post #38 of 52
I can't access any of WuWei's links.
post #39 of 52
Any updates?
post #40 of 52
I was wondering about an update too. I know this thread is a million years old, but I was in the same DDC with Periwinkle many years ago and I found myself wondering what ended up happening. Want to come back and let us know?
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