Lets look at his citations more clearly
(28) ATSDR web page "Public Health Statement for DDT, DDE, and DDD,"September 2002, Section 184.108.40.206 at http://www.atsdr.cdc.gov/toxprofiles/tp35.pdf
The exact wording that was paraphrased here was, "The proper development of many systems and functions depends on the timely action of hormones, particularly sex steroids; therefore, interfering with such actions can lead to a wide array of effects that may include altered metabolic, sexual, immune, and neurobehavioral functions. Effects of this type, that occur following exposure during fetal life via the placenta or early in life caused by either direct exposure to chemicals or exposure via maternal milk, are discussed in this section."
"DDT from the mother can enter her unborn baby through the placenta. DDT has been found in
amniotic fluid, human placentas, fetuses, and umbilical cord blood. DDT has been measured in
human milk; therefore, nursing infants are also exposed to DDT. In most cases, however, the
benefits of breast-feeding outweigh any risks from exposure to DDT in mother’s milk.
Nevertheless, women with unusually high amounts of DDT or metabolites in their bodies
(compared to background amounts measured in the general population) should be informed of
the potential exposure of the fetus if they become pregnant and the potential risks of breast-
Does not say anything about breast milk just a chemical fact sheet.
Does not mention the words breast, milk, maternal at all
(30) From http://www.niehs.nih.gov/health/docs/endocrine-disruptors-2010.pdf
page not found
(31) Committee on Developmental Toxicology, Board on Environmental Studies and Toxicology, in Scientific Frontiers in Developmental Toxicology and Risk Assessment (2000) , Commission on Life Sciences, The National Academies Press, p. 56
Found this, the only chapter that mentions breast milk is labled "Using model animals to assess and understand developmental toxicity."
(32) United States Office of Research May 25, 2001 Update, Environmental Protection and Development Agency, Dioxin: Scientific Highlights from Draft Reassessment (2000)
My pdf viewer wont let me look at this can someone else try?
(33) Male Reproductive Health and Environmental Xenoestrogens Jorma Toppari,et al. Environmental Health Perspectives - Vol 104, Supplement 4 - August 1996
A number of organochlorine pesticides
or pesticidal metabolites are found in
breast milk and human adipose tissue
). Several recent cross-sectional stud-
ies suggest a possible relationship between
levels of some organohalide residues in
human tissues and breast cancer risk,
although the observations are not entirely
consistent across studies, and no clear rela-
tionship has been established (23–30). Then also goes on to repeat the ddt article
(33a) Lower Serum Testosterone Associated with Elevated Polychlorinated Biphenyl Concentrations in Native American Men
Alexey Goncharov, et al., Environ Health Perspect. 2009 September; 117(9): 1454–1460. Published online 2009 May 20. doi: 10.1289/ehp.0800134 PMCID: PMC2737025 at http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2737025/
Akwesasne is a Native American (Mohawk) population of about 12,000 people residing along the St. Lawrence River in the Mohawk Territory at Akwesasne, near the junction of New York, Ontario, and Quebec. The territory is immediately downstream from three aluminum foundries, all of which used PCBs (Aroclor 1248) as hydraulic fluids, which leaked into the St. Lawrence River and its tributaries and have contaminated the local fish (Lacetti 1993). The Mohawk population is particularly vulnerable to PCB exposure because of their cultural and historical dependence on local fish, mammals, and waterfowl for food. Although their serum levels of PCBs are only moderately elevated [average, 5.29 ppb in males and 3.97 ppb in females (DeCaprio et al. 2005)], these values exceed the levels in persons without unusual exposure, which range from 0.9 ppb to 1.5 ppb (Agency for Toxic Substances and Disease Registry 2000). PCB levels in Mohawk breast milk (Hwang et al. 2001) and in serum (Fitzgerald et al. 2004) have been positively correlated with rates of consumption of local fish, although fish consumption has declined in recent years after consumption advisories were issued in the 1980s (Fitzgerald et al. 2004). Is the only mention of breast milk.
(34) ATDSR document on dioxins, section on environmental sources
The only example of this citation is on his websites. These words in this order do not appear anywhere else on the web.
(34a) National Academies of Science report on dioxins in the food supply. http://books.nap.edu/catalog.php?record_id=10763
(34b) http://www.epa.gov/iris/supdocs/dioxinv1sup.pdf in section 4.3.5, at end of that section, "...the resulting RfD in standard units is 7 × 10−10 mg/kg-day." In the EPA’s “Glossary of Health Effects”, RfD is defined: “RfD (oral reference dose): An estimate (with uncertainty spanning perhaps an order of magnitude) of a daily oral exposure of a chemical to the human population (including sensitive subpopulations) that is likely to be without risk of deleterious noncancer effects during a lifetime.”
(34c) U.S. EPA. Estimating Exposure To Dioxin-Like Compounds - Volume I: U.S. Environmental Protection Agency, Washington, D.C., EPA/600/8-88/005Ca., 2002, revised 2005 – http://cfpub.epa.gov/si/si_public_record_Report.cfm?dirEntryID=43870, Section II.6, "Highly Exposed Populations" (nursing infants are considered to be one of the highly-exposed populations), 4/94 (p. 39) "Using these procedures and assuming that an infant breast feeds for one year, has an average weight during this period of 10 kg, ingests 0.8 kg/d of breast milk and that the dioxin concentration in milk fat is 20 ppt of TEQ, the average daily dose to the infant over this period is predicted to be about 60 pg of TEQ/kg-d."
(34d) Schecter, A., et al. Chlorinated Dioxins and Dibenzofurans in Human Tissue from General Populations: A Selective Review, Environmental Health Perspectives Supplements 1994; 102(Supple 1): p. 159-171 and Schecter, A., et al. Congener-specific Levels of Dioxins and Dibenzofurans in U.S. Food and Estimated Daily Toxic Eequivalent Intake, Environmental Health Perspectives Journal 1994; 102(11): p. 962-966.
(34e) Chemosphere. 2007 Jan;66(2):311-9. Epub 2006 Jun 14. A comparison of PCDD/PCDFs exposure in infants via formula milk or breast milk feeding. Hsu JF, et al.
(35) "Technical Information for California Health Officials," May 2003, California Department of Health Services, Environmental Health Investigations Branch, p. 6
(36) Executive Summary: Assessment of the health risk of dioxins: re-evaluation of the Tolerable Daily Intake (TDI) WHO Consultation May 25-29 1998, Geneva, Switzerland, p. 27
(37) PCDDs, PCDFs, and PCBs concentrations in breast milk from two areas in Korea: body burden of mothers and implications for feeding infants, Jiyeon Yang et al. Chemosphere 46 (2002) 419–428
(37a) Infant Exposure to Dioxin-like Compounds in Breast Milk Lorber1 and Phillips2 VOLUME 110 | NUMBER 6 | June 2002 • Environmental Health Perspectives http://cfpub.epa.gov/ncea/cfm/recordisplay.cfm?deid=54708#Download
(37b) Transfer of Polycyclic Aromatic Hydrocarbons to Fetuses and Breast Milk of Rats Exposed to Diesel Exhaust, Tozuka, Watanabe et al., Kanazawa University and Tokyo Metropolitan Public Health Research Institute; Journal of Health Science 50(5) 2004 pp. 497-502
(38) ToxTown of National Library of Medicine, at http://toxtown.nlm.nih.gov/text_version/chemicals.php?id=69
(38a) Effects of developmental exposure to bisphenol A on brain and behavior in mice. Palanza P, et al., Environ Res. 2008 Oct;108(2):150-7. At http://www.ncbi.nlm.nih.gov/pubmed/18949834
(39) EPA/600/8-90/057F May 2002, Health Assessment Document for Diesel Engine Exhaust, Table 2-22. National Center for Environmental Assessment, Office of Research and Development, EPA
(40) Industrial Health 2000, 38, 259–268 Review Article: The Effects of Dioxin on Reproduction and Development Junzo YONEMOTO National Institute for Environmental Studies, Japan p. 262; Glorieux et al., 1988; Rovet et al., 1987; Haddow et al., 1999)." (Prioritization of Toxic Air Contaminants -- Children's Environmental Health Protection Act (State of California), October, 2001
(40a) C.A. Laroo et al., Emissions of PCDD/Fs, PCBs and PAHs from a Modern Diesel Engine Equipped with Catalyzed Emission Control Systerms, Environmental Science and technology, ACS Publications, June 30, 2011
(41) EPA/600/P-03/002F, November 2006: p. 11-28
(42) Reijnders, P.J. (1986) Reproductive failure in common seals feeding on fish from polluted coastal waters. Nature, 324, 456–457.
(43) from the ATSDR website page on Aroclors
(45) Ahlborg UG, Hanberg A, Kenne K. Risk Assessment of Polychlorinated Biphenyls (PCBs). Environmental Report in the Nord Series. Nord 26. Copenhagen: Nordic Council of Ministers, 1992.
(46) Environ Health. 2008; 7: 2. Published online 2008 January 17. doi: 10.1186/1476-069X-7-2 Rudel et al; licensee BioMed Central Ltd. PCB-containing wood floor finish is a likely source of elevated PCBs in residents' blood, household air and dust: a case study of exposure
(81b) Concentration of Persistent Organochlorine Compounds in the Placenta and Milk of the Same Women, Katarzyna Czaja et al., Ch. 21 of Persistent, Bioaccumulative, and Toxic Chemicals I, Robert L. Lipnick et al. editors, ACS Symposium Series, American Chemical Society, 2001; citing Jensen, A.A. et al, Chemical Contaminants in Human Milk, CRC Press, Inc., Boca Raton, Ann Arbor, Boston, 1991. Findings of above confirmed in animal tests, with even greater contrasts, in Ahlborg et al., Risk Assessment of Polychlorinated Biphenyls (PCBs), Nordic Council of Ministers, Copenhagen. Report NORD 1992; 26
(81c) U.S. EPA (2010) An exposure assessment of polybrominated diphenyl ethers. National Center for Environmental Assessment, Washington, DC; EPA/600/R-08/086F. online at http://www.epa.gov/ncea
(81d) Table 5-4 of above source
(81e) Section 5.6.2 of above source
(81f) Section 4.7 of above
(81g) Section 5.6.2 of above. The EPA states the figure as "44.1 ng/g lwt" (44.1 ng = 44,100 pg). For comparison purposes, the lipid (fat) weight indicated here needs to be converted to whole weight, which can be done as follows: The EPA here assumes a fat content of 4%. Using that figure, 44,100 pg/g lwt becomes 1760 pg/g wwt.
(82) National Academies Press: Health Risks from Dioxin and Related Compounds: Evaluation of the EPA Reassessment (2006), Board on Environmental Studies and Toxicology, National Academy of Sciences; the original source is not quoted directly because it is part of a draft, not for quoting
(82a) ATSDR Public Health Statement for Polychlorinated Biphenyls (PCBs), November 2000, Balfanz et al. 1993; MacLeod 1981; Wallace et al. 1996, p. 569
(82b) Pediatric Research (2001) 50, 331–336; doi:10.1203/00006450-200109000-00007 Early Childhood Determinants of Organochlorine Concentrations in School-Aged Children, Wilfried Karmaus et al.
(82c) Kommission “Human-Biomonitoring” des Umweltbundesamtes: Stoffmonographie PCB - Referenzwerte für Blut (Commission on Human Bio-Monitoring of the (German) Federal Environmental Office: Substance Monograph on PCB - - Reference Values for Blood) At http://www.umweltdaten.de/gesundheit/monitor/pcbblut.pdf , Section 8.3. found within http://www.umweltbundesamt.de/gesundheit/publikationen/index.htm , website of Umwelt Bundes Amt (German Federal Environmental Office). This article cited for this breastfed infant exposure data the source: Institut für Wasser-,Boden und Lufthygiene des Umweltbundesamtes, Kommission „Human-Biomonitoring“ des Umweltbundesamtes • Berlin: Referenzwerte für HCB,b-HCH, DDT und PCB in Frauenmilch (Institute for Water-, Soil and Air Hygiene of the Federal Environmental Office, Commission on Human Bio-Monitoring: "Reference Values for HCB,b-HCH, DDT und PCB in Human Milk." The text drawn on says, " "Die derzeit durchschnittlich vom Erwachsenen täglich aufgenommene Menge an PCB (ca. 0,02 μg PCB/kg KG ) liegt deutlich unter der ATD von 1 μg PCB/kg KG. Der gestillte Säugling erhält dagegen eine deutlich höhere PCB-Zufuhr (3 μg PCB/kg KG.", which Bing Translator translates as " "The amount taken daily average currently by the adults of PCB (approx. 0.02 μg PCB/kg bw ) is well below the ATD of 1 μg PCB/kg. The breastfed infant, however, receives a significantly higher PCB intake (3 μg PCB/kg bw."
(83) Infant Exposure to Dioxin-like Compounds in Breast Milk, Lorber and Phillips Volume 110 | Number 6 | June 2002 • Environmental Health Perspectives http://cfpub.epa.gov/ncea/cfm/recordisplay.cfm?deid=54708#Download Also EPA Home/Research/Environmental Assessment: An Evaluation of Infant Exposure to Dioxin-Like Compounds in Breast Milk, Matthew Lorber (National Center for Environmental Assessment, Office of Research and Development, U.S. Environmental Protection Agency) et al.
(84) In Prioritization of Toxic Air Contaminants - Children’s Environmental Health Protection Act, October, 2001: Dioxins
(85) Intake, fecal excretion, and body burden of polychlorinated dibenzo-p-dioxins and dibenzofurans in breast-fed and formula-fed infants. Abraham K, Knoll A, Ende M, Päpke O, Helge H. Children's Hospital, Virchow-Klinikum, Humboldt-Universität Berlin, Germany
(86) Regulatory Toxicology and Pharmacology, 37 (2003) 202 217 Dioxin risks in perspective: past, present, and future Hays and Aylward at http://acdrupal.evergreen.edu/envirohealth/system/files/Dioxin+risks+in+perspective.pdf
(87) Challenged Conceptions: Environmental Chemicals And Fertility" 2005, a publication of Stanford University School of Medicine, p. 4
(89) "Paying a Price for Loving Red Meat" in Personal Health, by Jane E Brody, New York Times: April 27, 2009
(89b) Brominated Flame Retardants, Third annual report to the Maine Legislature, Jan. 2007, Maine Dept. of Environmental Protection, Maine Center for Disease Control and Prevention, Dr. Deborah Rice et al.
(90) Shokuhin Eiseigaku Zasshi. 2004 Aug;45(4):175-83. PubMed – NCBI Polybrominated diphenyl ether flame retardants in foodstuffs and human milk. Akutsu K, Hori S. Osaka Prefectural Institute of Public Health: 1-3-69, Nakamichi,Osaka 537-0025, Japan
(90a) Zanieri L, Galvan P, Checchini L, Cincinelli A, Lepri L, Donzelli GP, et al. Polycyclic aromatic hydrocarbons (PAHs) in human milk from Italian women: influence of cigarette smoking and residential area. Chemosphere. 2007;67:1265–74., cited at http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3033466/
(90a1) Quotation to be found at http://www.scientificamerican.com/article.cfm?id=earth-talks-breast-feeding
(90a2) The wonder of breasts Florence Williams The Guardian, Friday 15 June 2012
(90b)Particle and Fibre Toxicology, Effects of prenatal exposure to diesel exhaust particles on postnatal development, behavior, genotoxicity and inflammation in mice. Karin S Hougaard et al., National Research Centre of the Working Environment, Copenhagen, Denmark. Published: 11 March 2008 Particle and Fibre Toxicology 2008, 5:3 doi:10.1186/1743-8977-5-3 This article is available from: http://www.particleandfibretoxicology.com/content/5/1/3
(91) CAS No: 7726-95-6) Health-based Reassessment of Administrative Occupational Exposure Limits, Committee on Updating of Occupational Exposure Limits, a committee of the Health Council of the Netherlands
(92) http://ehpnet1.niehs.nih.gov/docs/2001/109p75-88lakind/abstract.html Environmental Health Perspectives, Vol. 109, No.1, Jan. 2001
(93) Challenged Conceptions: Environmental Chemicals And Fertility" 2005, a publication of Stanford University School of Medicine, p. 10
(96) An Inventory of Sources and Environmental Releases of Dioxin-Like Compounds in the United States for the Years 1987, 1995, and 2000", EPA/600/P-03/002F, November 2006: especially Table 1-17. 2000 appears to be the most recent year for which the EPA provides national dioxin release data
(194) "Residential Proximity to Freeways and Autism in the CHARGE Study" , Environmental Health Perspectives, Published in 119(6) Jun 2011, Heather E. Volk, Irva Hertz-Picciotto et al., reported (with doubling comment) in ScienceDaily (Dec. 17, 2010), "Proximity to Freeway Associated With Autism"
(198) Figure 1-8 in EPA/600/P-03/002F November 2006 An Inventory of Sources and Environmental Releases of Dioxin-Like Compounds in the United States for the Years 1987, 1995, and 2000
(244) TOXICOLOGICAL PROFILE FOR LEAD, ATSDR August 2007, Section 3.3.2
(245) ATSDR Section 3, p. 264
(245b) U.S. EPA (2010) An exposure assessment of polybrominated diphenyl ethers. National Center for Environmental Assessment, Washington, DC; EPA/600/R-08/086F. online at http://www.epa.gov/ncea Executive Summary, p. xxiii
(254) EPA-452/R-97-006, December 1997, Table 2-3
Oh baby woke up.ill be back.
Edited by LLQ1011 - 7/21/13 at 5:04pm