I heard that it is not safe to have the amalgams removed while nursing because it stirs up the mercury and can put even more into the breastmilk.
post #61 of 306
1/19/06 at 2:36am
|So I guess if this is true, to truely reduce autism it might be a good idea to get your almagams removed, hair test & chelate yourself before you have children.. ain't hindsight grand????|
|MT, do you know by chance (well, actually by exhaustive research) how long the pill continues to affect one's system? I was on it from 14-21, and I went off it by choice when a nurse told me there were no long-term effects. Even then as unresearched as I was, I knew that that couldn't possibly be true.|
Ellen C G Grant
Scientific reasons for rejecting hormonal contraception
http://bmj.com/cgi/eletters/331/7527/1223-a#122773, 1 Dec 2005
Ellen C G Grant
Unreason in science - Irrational promotion of hormones as medications
http://bmj.com/cgi/eletters/330/7501/1214#107639, 23 May 2005
Ellen C G Grant
Pharmaceutical Medicine and the truth will out about hormonal contraceptives?
http://bmj.com/cgi/eletters/330/7496/0-g#106444, 11 May 2005
Ellen C G Grant
Reduction in mortality from breast cancer: Fall in use of hormones could have reduced breast cancer mortality
BMJ, Apr 2005; 330: 1024 ; doi:10.1136/bmj.330.7498.1024-a
...for screening, could be due to the reduction in the use of oral contraceptives and hormone replacement therapy after warnings of increased risk of thrombosis....
...Less than 12 months' use of most hormone replacement formulations increased the risk of breast cancer risk; quantified at 45-63% in the million...
Ellen C G Grant
Free lunches and hormone use promotions
http://bmj.com/cgi/eletters/330/7496/0-g#104469, 20 Apr 2005
Ellen C G Grant
Underreporting of congenital anomalies caused by hormonal contraceptives and nutritional deficiencies
http://bmj.com/cgi/eletters/330/7481/27#100397, 15 Mar 2005
Ellen C G Grant
Cancer, thrombosis and hormone use
http://bmj.com/cgi/eletters/330/7487/326-c#97526, 22 Feb 2005
Ellen C G Grant
Re: Re: Hormone use causes zinc deficiency and strokes
http://bmj.com/cgi/eletters/330/7487/342#97289, 20 Feb 2005
Ellen C G Grant
Hormone use causes zinc deficiency and strokes
http://bmj.com/cgi/eletters/330/7487/342#96999, 17 Feb 2005
Ellen C G Grant
Hormone use causes zinc deficiency
http://bmj.com/cgi/eletters/330/7487/342#96901, 15 Feb 2005
To the Editor,
In “Talking to patients” (Sept 5) Dr Adrian Morris confidently condemns the use of vitamins and minerals in the management of allergies. Dr Morris appears to have little knowledge of the advantages of using Nutritional Medicine to prevent or reduce allergic reactions to common foods and chemicals. The British Society for Allergy, Environmental and Nutritional Medicine (BSAENM) was founded 25 years ago and the Society’s textbook, “Environmental Medicine in Clinical Practice”, is an excellent introduction to this fundamental part of medical science.1
My own original research found that migraine and benign hypertension could be prevented by the avoidance of the major precipitants in the 1970s. Patients with higher serum copper/zinc ratios reacted to more foods. In several studies patients with low red cell magnesium levels were significantly more likely to have reactions. Low superoxide dismutase function in red cells, a sign of copper deficiency, also relates to increased reactivity. In my 45 years experience of Clinical Medicine and Research, these are the most important tests in diagnosing the reasons for many illnesses, including troublesome allergies, because deficiencies in mineral cofactors mean impaired function of hundreds of enzymes. Drug-Promoted-Medicine is trying to hold back the tide of progress towards understanding basic disease mechanisms. 2,3
Ellen C G Grant MBChB, DObstRCOG
1 Anthony H, Birtwhistle S, Eaton K, Maberley J. Environmental Medicine in Clinical Practice. BSAENM Publications 1997.
2 Gershwin M E, German J B, Keen C L. Nutrition and Immunology. Principals and Practice. Humana Press, Totowa, New Jersey, 2000
3.Clinical Nutrition of the Essential Trace Elements and Minerals. The Guide for Health Professionals. Humana Press, Totowa, New Jersey, 2000.
|But I didn't start being aware and eating well until I was 25, and really well until 28. I'm 34 now. Also, I have seven amalgams, which I am having removed this month.|
|Will extra selinium and vitamin C help with DS not being so affected by it?|
|Prof. John R. Arthur
The focus of my research is on the mechanisms whereby selenoproteins mediate the essential biological functions of selenium. Initial work on the role of selenium in iodothyronine deiodinases has led to the recognition that the micronutrient influences thyroid hormone and iodine metabolism as well as already widely studied antioxidant systems. This research has also concentrated on the role for selenium in redox regulation of cells as a component of thioredoxin reductase. I am investigating how selenium-induced changes in thioredoxin reductase activity may underlie changes in metabolism of lipids as well as oxidising and xenobiotic chemicals in the cell. These studies are part of our efforts to understand how low dietary selenium intake may contribute to the high incidence of coronary heart disease in Scotland (collaboration with Dr Geoff Beckett University of Edinburgh). I also intend to explore how selenium deficiency may be involved in control of cell transcription factors through redox regulation via thioredoxin. In collaboration with Prof. John Hesketh (University of Newcastle) studies are being carried out to identify mechanisms of enzyme/organ targeting of selenium to selenoproteins involved in antioxidant systems, redox mechanisms and thyroid hormone metabolism.
In projects funded by the Food Standards Agency (FSA) I am investigating the effects of selenium supplementation of adults with initially low selenium status on selenoprotein expression thyroid hormone metabolism and antioxidant status. This work is associated with further FSA funded studies to assess the influence of selenium status on immune function (collaboration with Professor M Jackson, University of Liverpool).
Arthur, J.R., Beckett, G.J. and Mitchell, J.H. (1999).The interactions between selenium and iodine deficiencies in man and animals. Nutrition Research Reviews 12, 57-75.
Arthur, J.R. and Beckett, G.J. (1999). Thyroid function. British Medical Bulletin 55, 658-668.
Brown, K.M., Pickard, K., Nicol, F., Beckett, G.J., Duthie, G.G. and Arthur, J.R. (2000). Effect of organic and inorganic selenium supplementation on selenoenzyme activity in blood lymphocytes, granulocytes platelets and erythrocytes. Clinical Science 98, 593-599.
Villar, D., Nicol, F., Arthur, J.R., Dicks, P., Cannavan, A., Kennedy, D.G. and Rhind, S.M. (2000). Type II and type III monodeiodinase activities in the skin of untreated and propylthiouracil-treated cashmere goats. Research in Veterinary Science 68, 119-123.
Arthur, J.R. (2000). The glutathione peroxidases. Cellular and Molecular Life Sciences 57, 1825-1835.
Arthur, J.R. and Beckett, G.J. (2000). Some biochemical functions of selenium in animals and man. In: Trace Elements in Man and Animals 10, Roussel, A.M., Anderson, R.A. and Favier, A.E. (eds.), Kluwer Academic/Plenum Publishers, New York, pp. 843-847.
Miller, S., Walker, S.W., Arthur, J.R., Nicol, F., Pickard, K., Lewin, M., Howie, A.F. and Beckett, G.J. (2001). Selenite protects human endothelial cells from oxidative damage by tertiary butyl hydroperoxide and induces thioredoxin reductase expression. Clinical Science 100 543-550.
Lewin, M.H., Hume, R., Howie, A.F., Richard, K., Arthur, J.R., Nicol, F., Walker, S.W. and Beckett, G.J. (2001). Thioredoxin reductase and cytoplasmic glutathione peroxidase activity in human foetal and neonatal liver. BBA 1526 237-241.
|Biol Neonate. 2002 Aug;82(2):122-7. Related Articles, Links
Maternal selenium nutrition and neonatal immune system development.
Dylewski ML, Mastro AM, Picciano MF.
Department of Nutrition, Pennsylvania State University, University Park, PA, USA.
We evaluated the impact of dietary selenium intake on neonatal immune cell differentiation and function. A low selenium intake during pregnancy and lactation produced reductions in maternal plasma selenium (33%, p = 0.0001), milk selenium (36%, p = 0.001), and corresponding neonatal plasma selenium (47%, p = 0.008). Thymocytes from neonates receiving low-selenium milk showed an impaired activation in vitro (p = 0.001). The percentages of CD8 cytotoxic T cells (p = 0.03), CD2 T cells (p = 0.09), panB cells ( = 0.02), and natural killer cells (p = 0.07) were all decreased in neonates nursed by mothers fed a low-selenium diet. The results indicate that maternal selenium intake impacts neonatal selenium status which in turn influences the neonatal immune system development. Copyright 2002 S. Karger AG, Basel
PMID: 12169835 [PubMed - indexed for MEDLINE]
Muscle Nerve. 2003 Jun;27(6):662-8.
Skeletal muscle disorders associated with selenium deficiency in humans.
Chariot P, Bignani O.
Department of Pathology, Hopital Henri-Mondor, Creteil, France. email@example.com
Skeletal muscle disorders manifested by muscle pain, fatigue, proximal weakness, and serum creatine kinase (CK) elevation have been reported in patients with selenium deficiency. The object of this report was to review the conditions in which selenium deficiency is associated with human skeletal muscle disorders and to evaluate the importance of mitochondrial alterations in these disorders. A systematic literature review using the Medline database and Cochrane Library provided 38 relevant articles. The main conditions associated with selenium deficiency fell into three categories: (1) insufficient selenium intake in low soil-selenium areas; (2) parenteral or enteral nutrition, or malabsorption; and (3) chronic conditions associated with oxidative stress, such as chronic alcohol abuse and human immunodeficiency virus (HIV) infection. In low soil-selenium areas, reversibility of muscle symptoms was similar after selenium supplementation and placebo administration, suggesting a role for other factors in the development of disease. In parenteral or enteral nutrition, or malabsorption, muscle symptoms improved after selenium supplementation in 18 of 19 patients (median delay: 4 weeks). The reason that only a minority of selenium-deficient patients present with skeletal muscle disorders is unclear and is possibly related to cofactors, such as viral infections and drugs. Prospective studies of selenium-deficient myopathies would be useful in critically ill patients, alcohol abusers, and HIV-infected patients.
Originally Posted by JaneS
Will be madly reading tonight!
I want to add that Tylenol/Advil greatly reduces glutathione levels so add that to the standard toxic medical arsenal. (And vitamin C increases it.)
|I just think much is not known about breastfeeding and removal. I did bf 1x a day during beginning of removal and I shouldn't have really. It's just NOT worth the worry, trust me. Just another one in a long line of stupid things I've done! My holistic dentist (who is a clw bf'ing mom) said it was fine during bf'ing. My ND not. There was one study of the IAOMT protocol that showed no appreciable raise in mercury blood level while using this advanced protection protocol.
But another study did show breastmilk contained 8 times more than blood level of mercury generally. I think they are at the DAMS site: www.amalgam.org
Originally Posted by chasmyn
Apparently I now live in a Seninium deficient area, but until June of 2004, lived in an adequate area. The question is, how much of the produce I ate THERE was grown locally? I have no idea. At any rate, it looks as if selenium supplementation would be a good idea based on the sources you cited.
|And apparently amalgam removal isn't recommended for breastfeeding mamas. Maybe selinium supplementation would be sufficient to keep the effects at bay for my nursling?|
|I'm not entirely sure whih is the lesser of two evils at this point (need to do more research) - "safe" removal of amalgams now with selenium/vitamin C supplementation (DS is 5 months old, I intend to do CLW, so 2 more years of nursing at least), or keep them in and keep the supplementation, risking also continued dosing of leached mercury in the breastmilk?|
|When I first decided to do removal I thought "safe removal" meant no worries. I ought to know better, nothing is as it seems. Always do more research! I am learning that this is true no matter what I think I already know.|
|As always, MT, you provide much insight and thought-provoking information. Thank you so much for taking the time to offer all of these links for me (and others who find them valueable). You are BETTER than Google because you know what you're looking for.|
|My first son was born with a severe heart defect called Hypoplastic Left Heart system (severely underdeveloped left heart). Fatal - ALWAYS - if no intervention (surgical, sadly).|
|MY gut told me when he was born that a big part of his defect came from my being in a poorly ventilated office sitting next to a warehouse full of TOXIC solvents every day, breathing in those fumes all damn day. Not every day, just on the days that the did restoration of fire/flood damaged items, but as the office was poorly ventilated, did those fumes ever really leave? I propped open the door to the outside no matter how cold it was just so I could breathe some days.|
|The other part, my gut said, was me. I don't know how exactly, but reading lately, all of the information above and this whole thread, I can definitely see the connection. As far as the medical and scientific community is concerned, HLHS is "random" - they have no idea of the cause, and because of its infrequency, it is difficult to do thorough studies.|
|At the time I thought I was eating healthy (according to SAD, I was) - I wasn't. Partly organic, mostly unprocessed, but still, many things that now I'd not touch. My amalgams, diet, environment, possible vitamin and mineral deficiencies, depleted adrenals, possible thyroid issues, all of these things I believe were contributors.|
|Honestly mostly that damn breathing in of the toxic solvents, but I can see now where my own biological connection can be made. (Not to take on blame, because really, I didn't know - but to educate myself and possibly others for the future.)|
|I didn't know enough then - I had him vaxed with that compromised immune system - I KNOW it affected him.|
|Despite his never being sick in his 2 years and 8 months of life, I know it affected him. He had started to speak and then after one of his vaxes (I think MMR but I'm not sure), he stopped altogether. One day he was saying "apple", the next, he forgot almost all of his vocalizations except the very basics (mama, daddy, kitty). My gut told me it was the vaxes once I learned about the damage.|