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|Cord closure abruptly halts the placental supply of glucose to the brain (used in aerobic and anaerobic respiration); the neonatal liver (glycogen stores) must begin to maintain blood glucose levels. A major portion of the liver’s blood supply is from the hepatic portal vein that derives its blood from the mesenteric arteries. If the gut (and hence the liver) is not “copiously perfused,” hypoglycemia may result in a neonatal convulsion. Deficient perfusion of the liver may also be a factor in bilirubin excretion and “physiological” jaundice.|
|During the third stage of labor, transition from placental dependency to self-sufficiency in life support is well understood by most midwives, lay and professional; the term used is “transing.” Most physicians think (and are taught) that this physiological process is pathological. For the midwife-home-delivered baby, the pulsating cord is routinely allowed to close itself regardless of the condition of the child at birth. Few if any of these neonates need NICU admission; this is a strong indication that delayed cord clamping – transing - is not a routine cause of pathology (jaundice, polycythemia, hypervolemia, hyperviscosity).|
Jen Mama of 2 precious boys (9) (6) and still in with my Matt after 12 years together.
Domestic Violence Children's Advocate and Counselor
Homeschooling mama to 6 year old DD.
Originally Posted by boscopup
No pitocin, had lots of jaundice, but DS was a preemie, so I think that affected his bilirubin levels more than anything else.